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Listar por autor "Casañas, Juan José"
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Artículo
Active zones and the readily releasable pool of synaptic vesicles at the neuromuscular junction of the mouse
Ruiz Laza, Rocío; Cano, Raquel; Casañas, Juan José; Gaffield, Michael A.; Betz, William J.; Tabares, Lucía (SOC NEUROSCIENCE, 2011-02-09)Synchronous neurotransmitter releaseis a highly regulated processthattakes place at specializations atthe presynapticmembrane ...
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Altered Intracellular Ca2+ Homeostasis in Nerve Terminals of Severe Spinal Muscular Atrophy Mice
Ruiz Laza, Rocío; Casañas, Juan José; Torres-Benito, Laura; Cano, Raquel; Tabares, Lucía (SOC NEUROSCIENCE, 2010)Low levels of survival motor neuron (SMN) protein result in spinal muscular atrophy (SMA), a severe genetic disease ...
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Prenatal treatment with rapamycin restores enhanced hippocampal mGluR‑LTD and mushroom spine size in a Down’s syndrome mouse model
Urbano Gámez, Jesús David; Casañas, Juan José; Benito, Itziar; Montesinos, María Luz (BioMed Central Ltd, 2021)Down syndrome (DS) is the most frequent genetic cause of intellectual disability including hippocampal-dependent memory ...
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Proteomic characterization of spinal cord synaptoneurosomes from Tg-SOD1/G93A mice supports a role for MNK1 and local translation in the early stages of amyotrophic lateral sclerosis
Casañas, Juan José; Montesinos Gutiérrez, María Luz (Elsevier, 2022-11)The isolation of synaptoneurosomes (SNs) represents a useful means to study synaptic events. However, the size and density ...
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Artículo
Rapamycin restores BDNF-LTP and the persistence of long-term memory in a model of Down's syndrome
Andrade-Talavera, Yuniesky; Benito, Itziar; Casañas, Juan José; Rodríguez Moreno, Antonio; Montesinos Gutiérrez, María Luz (Elsevier, 2015)Down's syndrome (DS) is the most prevalent genetic intellectual disability. Memory deficits significantly contribute to ...